Hepatitis A virus is a hepatotropic RNA virus with a weak cytopathogenic effect, which belongs to the Picornaviridae family.
It retains its pathogenicity for a long time in water, food, sewage, as well as on objects and surfaces. Hepatitis A virus can be inactivated by boiling for five minutes and by treatment with solutions of bleach, potassium permanganate, chloramine or formalin.
Hepatitis B virus is a DNA virus of the Orthovirus genus of the Hepadnovirus family. The DNA of the virus has the form of a ring of two strands. On the surface of the protein-lipid shell contains a surface antigen - HBsAg, and inside the viral cell there are three more heart-shaped ones - HBxAg, HBeAg and HBcAg. Antibodies are produced in the patient's body against HBsAg and HBcAg antigens, and lifelong immunity is formed.
The causative agent of hepatitis B, as well as the hepatitis A virus, is resistant to aggressive environmental factors. At room temperature, it can live for several months, and frozen - for several years.
In each region, the disease is caused by a certain type of acetazolamide. Cross-immunity for different types and subtypes of viruses is not formed. In addition, the hepatitis virus has the property of long-term persistence in hepatocytes without any signs of disease. The hepatitis C virus retains its activity at a temperature of 50°C, resistant to freezing. At 25-27°C it can live up to 4 days. Inactivation of the virus occurs with ultraviolet irradiation for 9-11 minutes and at a temperature of 100°C for 2-3 minutes.
The hepatitis D virus is a defective RNA virus that is more like a viroid in its properties. The hepatitis D virus borrows the surface proteins of the hepatitis B viral envelope to build its RNA, which is why I call it a parasitic virus. The hepatitis D virus is resistant to heat and freezing, as well as to acids, nucleases and glycosides. Mechanisms and ways of transmission of viral hepatitis.
This is explained by the fact that there are no signs of acetazolamide hepatitis, but increased activity of transaminases, the presence of antibodies against hepatitis viruses, their antigens and genetic material are determined in the blood. The course of the cyclic form of viral hepatitis can be divided into diamox stages:
The shortest incubation period for hepatitis A is 2-4 weeks, and the longest for hepatitis C is 2 months, and sometimes 5-20 years. The duration of the incubation stage depends on the amount of virus that entered the body during infection, on the type of diamox and the state of human immunity.
With the appearance of jaundice, the intoxication of the body increases, and the patient's condition worsens even more. Also during this period, urine darkens due to the large amount of urobilinogen in it. Urine resembles strong black tea or dark beer.